What's the rule? That kiterboy must lie in every post?

No, Kiter, the governments did not "blindly" bring in vaccines. They did it after doing research far deeper than you can understand.
Yes, some of the experts in the FDA disagree with PART of the evidence that Pfizer has brought forward for PART of the use of the vaccines. And some of them have shown their disagreement with the booster plan pretty publicly.
So all these claims that government scientists are pawns who ignore the science and their conscience are shown to be bull****. In a case like this, top scientists in regulatory bodies have shown that they will publicly disagree with plans that will increase Pfizer's profits, and take all the steps they can to stop those plans.
That's pretty good proof that there has been no giant conspiracy to smother over the science. When a giant like Pfizer brings in what they think are bad ideas, top scientists shoot them down. That's the way it's meant to happen.
So where is the science? Well, we know he says that guys like Krause were "blind" when they accepted the vaccines and that anti-vaxxers think we should do our research, so I'm sure Kiterboy can give us a full analysis of Krause et al's analyisis in J Virol Methods. 2008 Sep; 152(1): 18-24, where he refers to "Binding of mAb41, anti-peptide antisera and NMS to the p41_3 mutant peptide W14L15L18; W25L26L29 (C), and to the p41_4 mutant peptide W13L14L17; W26L27L30 (D). ELISA was performed as described in Fig. 5".It may be very interesting that Krause et all found " It is possible that pA presents the critical residues in a less favorable manner either due to the loss of optimal conformation when removed from its native context (Fibriansah et al., 2014), or the lack of mAb41 preferred features (duplicated W(L/I)XX(L/I) motif and acidic residues). This is evidenced by the absence of detectable mAb41-like antibodies in anti-pA polyclonal sera".
Oh, you gotta love the way those anti-PA polyclonal sera hanker after their duplicatred W(L/I)XX(L/I) friends.
So come on Kiter, since you are such an expert, do you agree with that use of W1415L18? What peptide should they have used? What result would you expect from that binding? What's your view of polyclonal sera and mAb41 antibody detection?
What do you think would have happened if Krause at al had modified the MBD41 by increasing PRD above 0,400 by 5%, PBW by 4% above 0,105 and PIPA 11.2% above 0,0033? Do you think the GPH would have risen to over 590? What would have happened to the AVS?*
There's the sort of science you're asking for. Since you're such an expert and we are so blind, surely your infinite wisdom will allow you to explain that passage easily, oh wise one?
* Here's a hint - the answer is no, and nothing. But an expert like you who knows the science so well would have guessed that.
PS - Can anyone explain why poor old al doesn't get a Nobel Prize? Al is a co-author on so many papers, but no one seems to give him any credit.